myoneural$51380$ - ορισμός. Τι είναι το myoneural$51380$
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Τι (ποιος) είναι myoneural$51380$ - ορισμός

MEDICAL CONDITION CAUSING BACK PAIN
Tension Myositis Syndrome; Tension myoneural syndrome

neuromuscular         
  • 3=[[Ligand]] (such as [[acetylcholine]])}} When ligands bind to the receptor, the [[ion channel]] portion of the receptor opens, allowing ions to pass across the [[cell membrane]].
  • thumb
  • At the neuromuscular junction, the nerve fiber is able to transmit a signal to the muscle fiber by releasing ACh (and other substances), causing muscle contraction.
  • Muscles will contract or relax when they receive signals from the nervous system. The neuromuscular junction is the site of the signal exchange. The steps of this process in vertebrates occur as follows:(1) The action potential reaches the axon terminal. (2) Voltage-dependent calcium gates open, allowing calcium to enter the axon terminal. (3) Neurotransmitter vesicles fuse with the presynaptic membrane and ACh is released into the synaptic cleft via exocytosis. (4) ACh binds to postsynaptic receptors on the sarcolemma. (5) This binding causes ion channels to open and allows sodium and other cations to flow across the membrane into the muscle cell. (6) The flow of sodium ions across the membrane into and potassium ions out of the muscle cell generates an action potential which travels to the myofibril and results in muscle contraction.Labels:A: Motor Neuron AxonB: Axon TerminalC. Synaptic CleftD. Muscle CellE. Part of a Myofibril
  • Motor Endplate
JUNCTION BETWEEN THE AXON OF A MOTOR NEURON AND A MUSCLE FIBER
Nueromuscular junction; Motor end plate; Motor end-plate; Neuromuscular junctions; Neuromuscular; Motor endplate; Neuromuscular Junction; Neuromuscular junction diseases; Neuromuscular agents; Myoneural junction; Myoneural; Neuromuscular plate; Neuromuscular transmission; End plate; Skeletal muscle junction; Neuromuscular block; Motor end organ; Nerve-Muscle Interface; Muscular block; Bromage score; Neuromuscular system; Neuromuscular synapse
¦ adjective relating to nerves and muscles.
Neuromuscular         
  • 3=[[Ligand]] (such as [[acetylcholine]])}} When ligands bind to the receptor, the [[ion channel]] portion of the receptor opens, allowing ions to pass across the [[cell membrane]].
  • thumb
  • At the neuromuscular junction, the nerve fiber is able to transmit a signal to the muscle fiber by releasing ACh (and other substances), causing muscle contraction.
  • Muscles will contract or relax when they receive signals from the nervous system. The neuromuscular junction is the site of the signal exchange. The steps of this process in vertebrates occur as follows:(1) The action potential reaches the axon terminal. (2) Voltage-dependent calcium gates open, allowing calcium to enter the axon terminal. (3) Neurotransmitter vesicles fuse with the presynaptic membrane and ACh is released into the synaptic cleft via exocytosis. (4) ACh binds to postsynaptic receptors on the sarcolemma. (5) This binding causes ion channels to open and allows sodium and other cations to flow across the membrane into the muscle cell. (6) The flow of sodium ions across the membrane into and potassium ions out of the muscle cell generates an action potential which travels to the myofibril and results in muscle contraction.Labels:A: Motor Neuron AxonB: Axon TerminalC. Synaptic CleftD. Muscle CellE. Part of a Myofibril
  • Motor Endplate
JUNCTION BETWEEN THE AXON OF A MOTOR NEURON AND A MUSCLE FIBER
Nueromuscular junction; Motor end plate; Motor end-plate; Neuromuscular junctions; Neuromuscular; Motor endplate; Neuromuscular Junction; Neuromuscular junction diseases; Neuromuscular agents; Myoneural junction; Myoneural; Neuromuscular plate; Neuromuscular transmission; End plate; Skeletal muscle junction; Neuromuscular block; Motor end organ; Nerve-Muscle Interface; Muscular block; Bromage score; Neuromuscular system; Neuromuscular synapse
Tension myositis syndrome         
Tension myositis syndrome (TMS), also known as tension myoneural syndrome or mindbody syndrome, is a name given by John E. Sarno to a condition of [musculoskeletal and nerve symptoms, most notably back pain].

Βικιπαίδεια

Tension myositis syndrome

Tension myositis syndrome (TMS), also known as tension myoneural syndrome or mindbody syndrome, is a name given by John E. Sarno to a condition of psychogenic musculoskeletal and nerve symptoms, most notably back pain. Sarno described TMS in four books, and stated that the condition may be involved in other pain disorders as well. The treatment protocol for TMS includes education, writing about emotional issues, resumption of a normal lifestyle and, for some patients, support meetings and/or psychotherapy. In 2007, David Schechter (a medical doctor and former student and research assistant of Sarno's) published a peer-reviewed study of TMS treatment in the journal "Alternative Therapies in Health and Medicine," showing a 54% success rate for chronic back pain. In terms of statistical significance and success rate, the study outperformed similar studies of other psychological interventions for chronic back pain.

The TMS diagnosis and treatment protocol are not accepted by the mainstream medical community. However, TMS and Sarno's treatment methods have received national attention, including a segment on ABC's 20/20; an episode of Larry King Live; an interview with Medscape; and articles in Newsweek, The Seattle Times, and The New York Times. Celebrity doctors who support TMS treatment include Andrew Weil and Mehmet Oz. Notable patients treated for tension myositis syndrome include Senator Tom Harkin, John Stossel, Howard Stern, and Anne Bancroft. In 2017, TMS was covered favorably in journalist C. J. Ramin's book Crooked. Ramin, who herself suffered from back pain for decades, criticized many of the popular back pain treatments and called Sarno "the rock star of the back world."